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doi: 10.1002/jso.24287 105 Di MontaGCaracCSimeoneEGrimaldiAMMaroneUDi MarzoMet al

One explanation offered for the discrepancy between its analgesic-antipyretic and anti inflammatory actions is its inability to inhibit COX in the presence of peroxides which a regenerated at sites of inflammation, but are not present in the brain

Excessive ROS/NO molecules cause intracellular calcium influxes (3) As a results imbalance the cellular membrane homeostasis and activate the stress response MAPK signaling pathway with its downstream signaling protein and mitochondrial caspase protein (4) that collapse the anti-oxidant mechanisms and induced the inflammatory cytokine secretion